• Login
    View Item 
    •   DSpace Home
    • SUNY Cobleskill
    • Student Publications
    • Student Research Showcase and SURC
    • Student Research Showcase - May 3, 2018
    • View Item
    •   DSpace Home
    • SUNY Cobleskill
    • Student Publications
    • Student Research Showcase and SURC
    • Student Research Showcase - May 3, 2018
    • View Item
    JavaScript is disabled for your browser. Some features of this site may not work without it.

    Browse

    All of DSpaceCommunities & CollectionsBy Issue DateAuthorsTitlesSubjectsThis CollectionBy Issue DateAuthorsTitlesSubjects

    My Account

    LoginRegister

    A Review of Cortisol: The “Stress Hormone”

    Thumbnail
    View/Open
    A Review of Cortisol.pdf (6.678Mb)
    Date
    2018-05
    Author
    Keller, Katelyn
    Baldwin, Sarah
    Brabetz, Barbara
    Metadata
    Show full item record
    Abstract
    Cortisol, also known as hydrocortisone, is a steroid hormone produced by the adrenal cortex in the adrenal gland of mammals. The zona fasciculata is the tissue that produces this glucocorticoid in response to stimulation by Adrenocorticotropic Hormone (ACTH). Cortisol is classified as a glucocorticoid, which aids in metabolism of fats, proteins, and carbohydrates. Cortisol is commonly referred to as the stress hormone because it is involved in the body’s natural response to stress. Most cells in the body contain receptors for cortisol, allowing it to have many functions within the body [5]. In normal cortisol release, its actions restore homeostasis after stress. Cortisol is responsible for controlling the body’s blood sugar levels, regulating metabolism. It acts as an anti-inflammatory and can promote the breakdown of glycogen, lipids, and proteins which reduces protein levels in most body cells. Cortisol influences memory formation, controls salt and water balance, and helps development of the fetus [1]. Cortisol is responsible for triggering key processes during parturition in many species. Cortisol is produced by specific enzymes known as the P450 enzymes, which are located on chromosome ten, region CYP17. 11-beta HSD1 and 11-beta HSD2 are the enzymes associated with the metabolism of cortisol [8]. The over or underproduction of cortisol can produce diseases within the body. Primary Hypercortisolism occurs when excessive levels of cortisol are present in the blood resulting in Cushing’s syndrome. Hypocortisolism or adrenal insufficiency results when the adrenal glands do not produce enough cortisol for the body. Addison’s disease refers to primary adrenal insufficiency, where the adrenal glands malfunction [1].
    URI
    http://hdl.handle.net/1951/70287
    Collections
    • Student Research Showcase - May 3, 2018 [15]

    SUNY Digital Repository Support
    DSpace software copyright © 2002-2022  DuraSpace
    Contact Us | Send Feedback
    DSpace Express is a service operated by 
    Atmire NV
     

     


    SUNY Digital Repository Support
    DSpace software copyright © 2002-2022  DuraSpace
    Contact Us | Send Feedback
    DSpace Express is a service operated by 
    Atmire NV